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Amlosac (Atenolol)

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Amlosac is a good medication for controlling chest pain (angina). It can also lower the chance of death if taken after a heart attack. Though Amlosac can be taken for blood pressure, it is not preferred and is typically only used if other blood pressure medications are not working.

Other names for this medication:
Aglodepin, Aloten, Amcard, Amloat, Amlocard, Amlokos, Amlopin, Amlothens, Amlovas, Amlovik, Ampin, Amvik, Atcom, Atemide, Atemos, Atenodip, Atenolol, Atpartc, Beten, Bibidip, Biduten, Calock, Cardenol, Cardipin, Cardipress, Espil, Gericard, Glipin, Joglit, Lakten, Licopin, Lodol, Malodip, Opime, Pdnol, Syten, Tecard, Teenol, Tefifin, Telol, Tenil, Tenocard, Tenofed, Tenopress, Tensicard, Tensimin, Vascodil

Similar Products:
Ambien, Adipex, Precose, Noroxin, Cytoxan, Calan, Carvetrend, Amodep, Trixide, Algyfree, Aziwok, Emfor,

Also known as: Atenolol


Amlosac is created by pharmacy specialists to combat angina, heart attack, high blood pressure, anxiety attacks and migraine. Target of Amlosac is to influence circulation. Amlosac is also known as Atenolol, Hipres, Atendol, Altol, Atelol, Antipressan, Atenix, Ateno, Betacard, Blokium, Catenol, Corotenol, Notenol, Oraday. Amlosac acts as an anti-angina remedy. Amlosac operates by controlling and decreasing the strength of heart contractions and, thus, influencing circulation. Amlosac is a beta-blocker. You can use Amlosac with other anti-high blood pressure medication. Generic name of Amlosac is Atenolol. Brand name of Amlosac is Amlosac.


Usual Adult Dose of Amlosac for Hypertension:

Initial dose: 50 mg orally once a day Maintenance dose: 50 to 100 mg orally once a day Maximum dose: 100 mg per day Comments: -If desired response not achieved after 1 to 2 weeks, increase to 100 mg may be beneficial. -Doses greater than 100 mg once a day did not result in significant additional antihypertensive effects. Use: For the treatment of hypertension alone or in combination with other antihypertensive agents.

Usual Adult Dose of Amlosac for Angina Pectoris Prophylaxis:

Initial dose: 50 mg orally once a day -Increase to 100 mg orally once a day after 1 week if optimal response not achieved Maintenance dose: 50 to 200 mg orally once a day Maximum dose: 200 mg per day Comments: -Some patients may require 200 mg per day to attain optimal effect. Use: For the long-term management of angina pectoris due to coronary atherosclerosis.

Usual Adult Dose for Angina Pectoris:

Initial dose: 50 mg orally once a day -Increase to 100 mg orally once a day after 1 week if optimal response not achieved Maintenance dose: 50 to 200 mg orally once a day Maximum dose: 200 mg per day Comments: -Some patients may require 200 mg per day to attain optimal effect. Use: For the long-term management of angina pectoris due to coronary atherosclerosis.

Usual Adult Dose of Amlosac for Myocardial Infarction:

50 mg orally twice a day or 100 mg orally once a day Comments: -If IV beta blockers are contraindicated or inappropriate, oral therapy should continue for at least 7 days post-myocardial infarction (MI). -Treatment with beta blockers post MI should generally continue for 1 to 3 years if there are no contraindications. Use: For the management of hemodynamically stable patients with definite or suspected acute myocardial infarction to reduce cardiovascular mortality.

Usual Geriatric Dose for Hypertension:

Initial dose: Consider reducing the starting dose to 25 mg orally once a day

Detailed Amlosac dosage information


Seek emergency medical attention or call the Poison Help line at 1-800-222-1222.

Overdose symptoms may include extreme weakness or lack of energy, very slow heart rate, shortness of breath, or fainting.


Store at room temperature between 20 and 25 degrees C (68 and 77 degrees F) away from moisture and heat. Keep container tightly closed. Throw away any unused medicine after the expiration date. Keep out of the reach of children.

Side effects

The most common side effects associated with Amlosac are:

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Side effect occurrence does not only depend on medication you are taking, but also on your overall health and other factors.


You should not use Amlosac if you are allergic to atenolol, or if you have:

a serious heart condition such as "AV block" (second or third degree); very slow heartbeats; or heart failure.

To make sure Amlosac is safe for you, tell your doctor if you have:

congestive heart failure; coronary artery disease (hardened arteries); asthma, bronchitis, emphysema; diabetes; overactive thyroid; liver or kidney disease; pheochromocytoma (tumor of the adrenal gland); peripheral vascular disease such as Raynaud's syndrome; or allergies (or if you are undergoing allergy treatments or skin-testing).

Using Amlosac during pregnancy could harm the unborn baby. Tell your doctor if you are pregnant or if you become pregnant while using this medicine.

Atenolol can pass into breast milk and may harm a nursing baby. Tell your doctor if you are breast-feeding a baby.

Amlosac is not approved for use by anyone younger than 18 years old.

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Inflammatory breast cancer-comparing the effectivity of preoperative docetaxel-epirubicine protocol to conventional antracycline-containing chemotherapy to achieve clinical benefit and complete pathological response. Patients were stratified into tertiles according to the number of procedures performed by their surgeons and at their hospital. In contrast, there was a significant relationship between attained LDL-C on statin therapy and TT and FT levels. The ubiquitous presence of the digital camera and digital technology has made it extremely easy and affordable to take high-quality images. Diagnostic value of overall estimation of myocardial ischemia in patients with ischemic heart disease from EKG monitor computer analysis during the treadmill test. In the primary prevention of cardiovascular disease, proper risk stratification must be carried out with specific attention given to lifestyle changes. The long-term use of antiplatelet agents is a key component of secondary prevention measures following acute coronary syndromes, including ST-segment elevation myocardial infarction. Sequencing of intron 3 of HMGA2 uncovers the existence of a novel exon. Ultrastructural studies of the myenteric plexus and smooth muscle in organotypic cultures of the guinea-pig small intestine. If this is the case, then one would expect conditioned taste avoidance not to be disrupted if the lPBN is functional during taste processing but not during LiCl processing.

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Total hip arthroplasty in patients younger than 30 years of age following developmental dysplasia of hip (DDH) in infancy. After induction of pancreatitis immunolabelling still demonstrated a colocalisation of cathepsin B and trypsinogen in secretory granules and newly formed Golgi-derived secretory vacuoles. Large gantry openings easily accommodate patients in a sitting or semiprone position without the need for attaching a seat device to the table top. Conversely, PHTN-FTR is determined by valvular tethering with tenting linked to RV elongation and elliptical/spherical deformation. Intermittent claudication--surgical reconstruction or physical training?. When the flanking residues are included in the calculation the hepapeptide P-VNTFV-H (ribonuclease) again yields an ensemble of beta-strands. Lipid length controls antigen entry into endosomal and nonendosomal pathways for CD1b presentation. Our TIRF microarray system can be mounted on upright or inverted microscopes or interfaced directly with CCD cameras equipped with a single objective, facilitating the development of portable devices. Correlation between clinical characteristics, survival and genetic alterations in patients with hepatocellular carcinoma from Saudi Arabia.

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Metallothioneins (MTs) constitute a class of low molecular weight, cysteine-rich, metal binding proteins which are regulated at the level of gene transcription in response to heavy metals and other adverse treatments. The analysis of these diagrams provides useful information allowing: (a) the determination of the most important primitives for a given orbital, (b) the core-valence separation, and (c) support for the development of contracted basis sets by the segmented method. The construction and analysis of MFCs requires knowledge of different scientific and engineering fields, ranging from microbiology and electrochemistry to materials and environmental engineering. Overall, the sequence and organization of the CETP gene do not resemble those of other lipid-metabolizing enzymes or apolipoproteins. Intriguingly, we demonstrated that the promoter of the TAC1 gene must act in synergy with a remote enhancer, identified using comparative genomics, to respond to MAPK signalling that modulates the expression of TAC1 in sensory neurones. Double half-cubane copper(II) complexes available from copper(II) 1,1,1,5,5,5-hexafluoropentane-2,4-dionate dissolved in formamides. Histopathological findings revealed mild focal cortical dysplasia.

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Distribution and morphology of transgenic mouse oligodendroglial-lineage cells following transplantation into normal and myelin-deficient rat CNS. The liver veins were significantly wider in the patients, and the widest veins were found in those with pleural effusion. Endothelium-dependent vasodilation is impaired in patients with congestive heart failure. Prediction of residues in discontinuous B-cell epitopes using protein 3D structures. Acoustic stimulation of bone marrow, as well as the isolation of hMSCs in the absence of fetal bovine serum, increased the osteogenic and ECM production potential of the cells, respectively. The Kas for native receptors, asialoreceptors and aglycoreceptors, are also comparable, being 2.0 x 10(9) M-1, 1.9 x 10(9) M-1 and 1.7 x 10(9) M-1 respectively. While transition metal oxides have been thoroughly investigated as coatings for electrochemical capacitors due to their pseudocapacitance, little work has been done investigating other oxide coatings. Although development leads unidirectionally toward more restricted cell fates, recent work in cellular reprogramming has proven that one cellular identity can strikingly convert into another, promising countless applications in biomedical research and paving the way for modeling diseases with patient-derived stem cells. A 30-year-old woman presented with a 1-year history of a palpable, asymptomatic right breast mass without axillary lymphadenopathy and family history of breast carcinoma. DDD is a common ailment with enormous medical, psychosocial, and economic ramifications.

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