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Allgout (Allopurinol)

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Allgout is used to prevent and treat gout. Gout is caused by too much uric acid in your body. When the level becomes too high, crystals may form in and around certain joints and your kidneys.

Other names for this medication:
Algoric, Allgout, Alopur, Aloric, Aloriv, Alpuric, Aluric, Axanthine, Ciploric, Estinol, Gudloric, Kayloric, Klorinol, Lodiric, Myloric, Purinol, Seloric, Swiloric, Ureka, Uriclear, Urigon, Urinol, Urxoall, Xoric, Zanupirol, Zeric, Zyloric, Zyrinol, Zytol

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Also known as: Allopurinol


Generic Allgout is used for treating gout caused by excessive levels of uric acid in the blood (hyperuricemia). Hyperuricemia occurs when the body produces more uric acid than it can eliminate. The uric acid forms crystals in joints (gouty arthritis) and tissues, causing inflammation and pain. Elevated blood uric acid levels also can cause kidney disease and stones. Generic Allgout prevents the production of uric acid by blocking the activity of the enzyme that converts purines to uric acid. Generic Allgout prevents the production of uric acid by blocking the activity of the enzyme that converts purines to uric acid. Allgout is also known as Allopurinol, Allohexal, Allosig, Progout, Zyloric, Puricos. Generic name of Generic Allgout is Allopurinol. Brand names of Generic Allgout are Allgout, Aloprim.


Take this medicine exactly as directed by your doctor. Do not take more of it, do not take it more often, and do not take it for a longer time than your doctor ordered. To do so may increase the chance of side effects.

You may take this medicine after meals to avoid stomach upset.

Take this medicine with plenty of liquids to help prevent kidney stones. Check with your doctor about the amount of liquid you should drink each day. You should also increase your fiber intake.

You may need to lessen your intake of animal protein, sodium, refined sugars, or foods rich in oxalate and calcium, especially if you have kidney stones that keep coming back.

The dose of this medicine will be different for different patients. Follow your doctor's orders or the directions on the label. The following information includes only the average doses of this medicine. If your dose is different, do not change it unless your doctor tells you to do so.

The amount of medicine that you take depends on the strength of the medicine. Also, the number of doses you take each day, the time allowed between doses, and the length of time you take the medicine depend on the medical problem for which you are using the medicine.

For oral dosage form (tablets): For gout: Adults—At first, 100 to 300 milligrams (mg) per day, taken once a day or in divided doses. Your doctor may adjust your dose as needed. However, the dose is usually not more than 800 mg per day. Children—Use and dose must be determined by your doctor. For high uric acid levels caused by cancer medicines: Adults and children 11 years of age and older—600 to 800 milligrams (mg) per day, taken in divided doses for 2 to 3 days. Children 6 to 10 years of age—300 mg per day, taken once a day for 2 to 3 days. Children younger than 6 years of age—150 mg per day, taken once a day for 2 to 3 days. For kidney stones: Adults—200 to 300 milligrams (mg) per day, taken once a day or in divided doses. Your doctor may adjust your dose as needed. However, the dose is usually not more than 800 mg per day. Children—Use and dose must be determined by your doctor.


If you overdose Generic Allgout and you don't feel good you should visit your doctor or health care provider immediately.


Store at room temperature between 15 and 25 degrees C (59 and 77 degrees F) away from light and moisture. Do not store in the bathroom. Throw away any unused medicine after the expiration date. Keep out of the reach of children.

Side effects

The most common side effects associated with Allgout are:

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IKZF1 is expressed in early hematopoiesis, including precursor myeloid cells. Lentiviral vectors are promising tools for liver disease gene therapy, because they can achieve protracted expression of transgenes in hepatocytes. Comparison of bone mineral density between premenopausal and postmenopausal women in Bangladesh. Amides of cis and trans adamantanol showed good inhibitory activity against Syk as well as remarkable activity in LAD2 cells degranulation assay. Ligands noroxin 400 mg para que sirve located within a cholesterol domain enhance gene delivery to the target tissue. Application of random effects to the study of resource selection by animals. Conversely, restoring the integrity of the cranial vault structure can result in better dissipation of traumatic energy. We review the key diagnostic features that differentiate AU from other physical urticarias. Multiple DNA recognition sites by sequence-specific DNA-binding proteins in the region of replication initiation site located 5-end upstream to the chicken alpha-globin gene domain. The length of the transmembrane assemblies is determined both by the thermodynamics of the intermolecular interactions in the supermolecule and by the dimension and physical chemical properties of the bilayer.

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Risk associations between lipoprotein-associated phospholipase A2 (Lp-PLA2) and adverse outcomes in patients with coronary heart disease (CHD) remain unclear. Based on this analysis, we may conclude that the PASI is a reliable indicator of psoriasis severity because it shows significant concordance when independent evaluations are performed. Sublingual immunotherapy (SLIT) has also been shown to improve asthma symptoms and medication use. Extracellular matrix (ECM) secretion and osteogenic differentiation in periodontal ligament fibroblasts (PDLF) facilitate the neogenesis of alveolar bone, which micardis starting dose is the cellular basis for alveolar bone repair. Regulatory requirement for three dose levels in rodent genetic toxicity assays. A multilayered approach of Si/SiO to promote carrier transport in electroluminescence of Si nanocrystals. Medical records of horses presented for surgical treatment of sinonasal disease from 1996-2007 were reviewed. High quality MRI data can be acquired and analyzed from older adults in population studies, enhancing their capacity to examine imaging biomarkers in relation to cognitive aging and dementia. We identified common upregulated transcription factors, which might serve as a source for generating a cocktail of factors able to directly transdifferentiate somatic cells into HLCs. Single-cell sequencing of bone marrow epithelial-like cells, in parallel with intra-tumor genetic heterogeneity profiling from bulk DNA, is a powerful approach to identify and study DTCs, yielding insight into metastatic processes.

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Morphological study on the central subdivision of the rat hypothalamic ventromedial nucleus. Axons located at the periphery of a nerve bundle have lower thresholds and different excitation sites compared with axons located near the center of a nerve bundle. tigrinus are by far higher than those reported in other remediation studies conducted under unsaturated solid phase conditions on actual site soils historically contaminated by Aroclor 1260. Published recommendations suggest three visual field (VF) tests per year are required to identify rapid progression in a newly diagnosed glaucomatous patient over 2 years. To review the domains in which computerized information systems have proven beneficial in facilitating the metabolic and nutritional management. A total of 157 consecutive diabetic patients who had undergone CCTA were included retrospectively. Desmopressin, but not vasopressin, decreases activity of the hypothalamo-neurohypophysial system in Brattleboro rats.

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In a word, our data provide new insights into the global biological role of miR-103. Polyphenols probably act in such remedies by virtue of their astringent action and current views on the molecular basis of this property are outlined and discussed. Bandgap renormalization and work function tuning in MoSe2/hBN/Ru(0001) heterostructures. We present the development of an in vitro transcription system, composed of purified N4 RNA polymerase II and solubilized P17, which accurately utilizes in vivo sites of transcription initiation. Our study demonstrates the safety and potential benefit of sorafenib in reducing the incidence of hepatocellular carcinoma recurrence and in extending disease-free and overall survival for high-risk liver transplant recipients.

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